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Antibiotics degradation by advanced oxidation process (AOPs): Recent advances in ecotoxicity and antibiotic-resistance genes induction of degradation products

  • Shuo Li
  • , Yanan Wu
  • , Heshan Zheng*
  • , Hongbin Li
  • , Yongjie Zheng
  • , Jun Nan
  • , Jun Ma
  • , Dillirani Nagarajan
  • , Jo Shu Chang*
  • *Corresponding author for this work
  • Qiqihar University
  • National Engineering Center of Urban Water Resources
  • School of Environment, Harbin Institute of Technology
  • National Cheng Kung University
  • Tunghai University
  • Yuan Ze University

Research output: Contribution to journalReview articlepeer-review

Abstract

Antibiotic contamination could cause serious risks of ecotoxicity and resistance gene induction. Advanced oxidation processes (AOPs) such as Fenton, photocatalysis, activated persulfate, electrochemistry and other AOPs technologies have been proven effective in the degradation of high-risk, refractory organic pollutants such as antibiotics. However, due to the limited mineralization ability, a large number of degradation intermediates will be produced in the oxidation process. The residual or undiscovered ecological risks of degradation products are potential safety hazards and problems necessitating comprehensive studies. In-depth investigations especially on the full assessments of ecotoxicity and resistance genes induction capability of antibiotic degradation products are important issues in reducing the environmental problems of antibiotics. Therefore, this review presents an overview of the current knowledge on the efficiency of different AOPs systems in reducing antibiotics toxicity and antibiotic resistance.

Original languageEnglish
Article number136977
JournalChemosphere
Volume311
DOIs
StatePublished - Jan 2023
Externally publishedYes

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

  1. SDG 3 - Good Health and Well-being
    SDG 3 Good Health and Well-being

Keywords

  • Advanced oxidation process
  • Antibiotic degradation
  • Antibiotic-resistance genes
  • Products toxicity

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